目的：探讨高强度运动后微高压氧干预对机体恢复及其后续无氧运动表现的影响。方法：采用随机交叉自身对照设计对11位被试进行2次干预实验，实验间隔设置一周洗脱期，以消除前一次干预的遗留效应；实验期间，被试完成2次温盖特最大无氧功率测试（WAnT 1和WAnT 2），测试间隔为60 min；测试间隔期间，被试随机接受60 min常压常氧干预（1.0 ATA，20.93% O₂）或微高压氧干预（1.3 ATA，40% O₂）；温盖特最大无氧功率测试后记录其峰值功率（P_P）、平均功率（P_M）、最大心率（HR_max）、峰值血乳酸浓度（BLa_peak）、经皮动脉血氧饱和度（SpO₂）及主观疲劳感觉评分，并在干预实验期间每10 min记录一次心率（HR）、血乳酸浓度（BLa）、经皮动脉血氧饱和度；干预结束后，使用主观疲劳感觉评分（PR）评估机体恢复状况；实验数据以均值±标准差（M±SD）表示，采用配对样本t检验比较组内差异；实验期间，各生理指标进行双因素重复测量方差分析，统计显著性水平设定为p< 0.05。结果：1）在常压常氧条件下，第2次温盖特最大无氧功率测试后的平均功率相较首次温盖特最大无氧功率测试后显著降低（p< 0.05, d = 0.51）；2）在微高压氧条件下，2次温盖特最大无氧功率测试后的平均功率均无显著变化（p = 0.83）；3）血乳酸浓度在微高压氧干预后与常压常氧干预后存在显著差异，经皮动脉血氧饱和度也有显著差异（p< 0.05）；4）微高压氧条件下的主观疲劳感觉评分与常压常氧条件下的测试结果存在显著差异（p< 0.05），但是运动后最大心率及干预期间心率在不同干预条件下均无显著差异（p> 0.05）。结论：高强度运动后进行微高压氧干预，可加速清除血乳酸，增加经皮动脉血氧饱和度，有效改善主观疲劳状态，进而有助于促进机体恢复，并维持后续无氧运动表现。

Objective: To investigate the effects of micro hyperbaric oxygen intervention on physiological recovery and subsequent anaerobic performance after high-intensity exercise. Methods: A randomized crossover design was used to conduct two intervention experiments in 11 participants, with a one-week washout period set between experiments to minimize potential carryovereffects from the previous intervention. During the experimental period, participants completed two Wingate Anaerobic Tests (WAnT 1 and 2) with a 60minute interval between tests. During this interval, they were randomly assigned to receive either normobaricnormoxia (NN: 1.0 ATA, 20.93% O₂) or microhyperbaric oxygen (MH: 1.3 ATA, 40% O₂) for 60 minutes. Following each WAnT, peak power (P_P), mean power (P_M), maximal heart rate (HR_max), peak blood lactate concentration (BLa_peak), percutaneous arterial oxygen saturation (SpO₂), and ratings of perceived exertionwere recorded. Throughout the intervention, heart rate (HR), blood lactate concentration (BLa), and SpO₂ were measured every 10 minutes. Additionally, at the end of the intervention, the perceived recovery (PR) scale was used to assess the physiological recovery status. The experimental data are expressed as mean ± standard deviation (M ± SD), and the paired-samples t-test was used to compare within-group differences. Temporal variations in physiological parameters during the intervention were analyzed using two-way repeated measures ANOVA (condition × time), and the statistical significance level was set at p< 0.05. Results: 1) Under normobaricnormoxic conditions, the mean power output during the second Wingate anaerobic test was significantly lower than that during the first Wingate test (p< 0.05, d = 0.51); 2) under micro-hyperbaric oxygen conditions, no significant difference in mean power output was observed between the two Wingate anaerobic tests (p = 0.83); 3) blood lactate concentration and percutaneous arterialoxygen saturation differed significantly between the micro-hyperbaric oxygen and normobaricnormoxic conditions following the intervention (p< 0.05); and 4) the rating of perceived fatigue was significantly lower under the micro-hyperbaric oxygen condition than under the normobaricnormoxic condition (p< 0.05), whereas no significant differences were observed in post-exercise maximal heart rate or heart rate during the intervention between the two conditions (p> 0.05).Conclusion: Micro-hyperbaric oxygen intervention following high-intensity exerciseaccelerate blood lactate clearance, increasepercutaneous arterial oxygen saturation, andeffectively alleviate subjective fatigue, thereby facilitatingphysiological recovery and helping maintain subsequent anaerobic performance.